The Fiber Disease

Human Anatomy, Physiology, and Medicine. Anything human!

Moderators: honeev, Leonid, amiradm, BioTeam

Nadas Moksha
Posts: 248
Joined: Thu Aug 10, 2006 7:41 am

Post by Nadas Moksha » Sun Nov 05, 2006 4:16 am

this may not be breaking news but the environment fits the time...
a sky over london will know where I am. ... 03/003.htm

Artificial Molecular Receptors

Another early goal of nanomedicine is to study how biological molecular receptors work, and then to build artificial binding sites on a made-to-order basis to achieve specific medical results. Buddy D. Ratner at the University of Washington in Seattle has researched the engineering of polymer surfaces containing arrays of artificial receptors. In a recent series of experiments, Ratner and his colleagues used a new radiofrequency-plasma glow-discharge process to imprint a polysaccharide-like film with nanometer-sized pits in the shape of such biologically useful protein molecules as albumin (the most common blood protein), fibrinogen (a clotting protein), lysozyme and ribonuclease (two important enzymes), and immunoglobulin (antibodies). Each protein type sticks only to a pit with the shape of that protein. Ratner's engineered surfaces may be used for quick biochemical separations and assays, as well in biosensors and chemosensors, because such surfaces will selectively absorb from solution only the specific protein whose complementary shape has been imprinted, and only at the specific place on the surface where the shape is imprinted. The RESIST Group at the Welsh School of Pharmacy at Cardiff University and others have looked at how molecularly imprinted polymers could be medically useful in clinical applications such as controlled drug release, drug monitoring devices, and biological and antibody receptor mimics.


Dendrimers represent yet another nanostructured material that may soon find its way into medical therapeutics. Starburst dendrimers are tree-shaped synthetic molecules with a regular branching structure emanating outward from a core. Dendrimers form nanometer by nanometer, so the number of synthetic steps or "generations" dictates the exact size of the particles in a batch. Each molecule is typically a few nanometers wide though some have been constructed up to 30 nanometers wide, incorporating more than 100,000 atoms. The peripheral layer of the dendrimer particle can be made to form a dense field of molecular groups that serve as hooks for attaching other useful molecules, such as DNA, which hunker down amongst the outermost branches.

In 1998, James R. Baker Jr. co-founded the Center for Biologic Nanotechnology at the University of Michigan to bring together doctors, medical researchers, chemists and engineers to pursue the use of dendrimers as a safer and more effective genetic therapy agent. For Baker, these nanostructures are attractive because they can sneak DNA into cells while avoiding triggering an immune response, unlike viral vectors commonly employed today for transfection. The dendrimer molecule is decorated with specific snippets of DNA, then injected into biological tissue. Upon encountering a living cell, dendrimers of a certain size trigger a process called endocytosis in which the cell's outermost membrane deforms into a tiny bubble, or vesicle. The vesicle encloses the dendrimer which is then admitted into the cell's interior. Once inside, the DNA is released and migrates to the nucleus where it becomes part of the cell's genome. The technique has been tested on a variety of mammalian cell types, with clinical human trials of dendrimer gene therapy originally scheduled to begin in 2001. Donald Tomalia, another co-founder of the Center for Biologic Nanotechnology, reported using glycodendrimer "nanodecoys" to trap and deactivate influenza virus particles. The glycodendrimers present a surface that mimics the sialic acid groups normally found in the mammalian cell membrane, causing virus particles to adhere to the outer branches of the decoys instead of the natural cells.

In July 2003, Starpharma was cleared by the U.S. FDA for human trials of their dendrimer-based anti-HIV product. Their product has been successful in preventing simian-HIV.

Smart Drugs

Medical nanomaterials also may include "smart drugs" that become medically active only in specific circumstances. A good example is provided by Yoshihisa Suzuki at Kyoto University, who has designed a novel drug molecule that releases antibiotic only in the presence of an infection. Suzuki started with the common antibiotic molecule gentamicin and bound it to a hydrogel using a newly developed peptide linker. The linker can be cleaved by a proteinase enzyme manufactured by Pseudomonas aeruginosa, a Gram-negative bacillus that causes inflammation and urinary tract infection, folliculitis, and otitis externa in humans. Tests on rats show that when the hydrogel is applied to a wound site, the antibiotic is not released if no P. aeruginosa bacteria are present. But if any bacteria of this type are present, then the proteolytic enzyme that the microbes naturally produce cleaves the linker and the gentamicin is released, killing the bacteria. "If the proteinase specific to each bacterium [species] can be used for the signal," writes Suzuki, "different spectra of antibiotics could be released from the same dressing material, depending on the strain of bacterium." This specificity of action is highly desirable because the indiscriminate prophylactic use of antibiotics is associated with the emergence of strains of drug-resistant bacteria, and most antibiotics apparently have at least some toxicity for human fibroblasts.

Immunotoxins are another class of smart drugs, in this case activating only in the presence of cancer cells. An immunotoxin molecule is an engineered hybrid of functional protein modules fabricated from two different types of proteins: a toxin and an antibody. Toxin proteins are normally produced and released by infectious bacteria. The protein binds to the surface of a host cell, penetrates it, and kills it. Toxin molecules are so potent that just a few of them can kill a cell. Antibodies are proteins produced by the immune system to recognize and bind to specific foreign materials. An immunotoxin molecule is made by fusing a part of the gene encoding a toxin with a part of the gene encoding an antibody that recognizes surface features on cancer cells. This creates a novel gene that can be used to express a new synthetic protein molecule. This new molecule will bind only to a cancer cell (via a module from the antibody protein), then penetrate it and kill it (via modules from the toxin protein). The first experiments with mice showed that these engineered proteins successfully eliminated certain tumors. Then early in 2000, National Cancer Institute researchers confirmed that an immunotoxin made from a truncated form of Pseudomonas exotoxin was cytotoxic to malignant B-cells taken from patients with hairy cell leukemia. A second clinic trial at the Universitaet zu Koeln in Germany also found that a ricin-based immunotoxin had moderate efficacy against Hodgkin's lymphoma in some patients.

------oooohhhh and TAM TAM, on that extra cellular marix.. . . . . .

"Our group is developing highly aligned ultrathin fiber bundles which mimic the natural fibrous structure in tissue through electrospinning for neuronal regeneration purposes, since it is generally believed that organized neuronal architecture is essential for nervous system
development, functioning, and regeneration [23–25].Adjusting process parameters and polymer solution characteristics can vary fiber sizes
and properties. Many polymers are adaptable for an electrospinning process, such as polyethylene oxide, poly(ethylene-co-vinyl alcohol), DNA, polyaramids, polycaprolactone,PLA, PGA, polyaniline, and polypeptides [21]. Electrospinning can be used to prepare bioac-
tive nanofibers, which may be used for carrying active biomolecules. For example, enzymes can be immobilized via material engineering and improved stability and activity of the enzymes were observed [26]. The electrospun structure, composed of fibers ranging from 50
to 1000 nm in diameter, features a morphologic similarity to the extracellular matrix (ECM)of natural tissue. A wide range of pore diameter distribution, high porosity, and effective mechanical properties could be obtained through the well-developed electrospinning tech-

and since im a dial-up snail i had to skip these pdfs...... . . . ... iaoLin.pdf.

please post any portions of inerest... . ,. .


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Post by Marcos » Sun Nov 05, 2006 4:31 am

Are the majority of the posters here Amish?

Nadas Moksha
Posts: 248
Joined: Thu Aug 10, 2006 7:41 am

Post by Nadas Moksha » Sun Nov 05, 2006 4:43 am

amish coke cartel of 150 murderous passions ...... . . . .
damn i cant shake the chronic fatigue.... . ..
but check where this is comming from ... . . .
Division of Agricultural Sciences and Natural Resources – FY 2004 Research Abstracts

The Biochemical Basis for Resistance of Cotton to Pathogens and Pests

Defense chemicals contribute to the pathogen and pest resistance of cotton, but make its seed toxic to nonruminants. Suppression of the biosynthetic pathway to these chemicals specifically in seeds may detoxify the seed while maintaining overall plant resistance. Genes in this pathway and others that are active during bacterial blight resistance are being sought. (1504)

Sponsors: National Science Foundation, Cotton Incorporated, USDA/ARS Southern Crops Research Laboratory, Oklahoma Agricultural Experiment Station

PIs: Margaret Essenberg, Margaret Pierce

Interactions of Bacterial Pathogens and Animal Hosts

Factors affecting survival of Brucella abortus in macrophages and other cells will be investigated by studying effects on survival in macrophages of loss of specific genes involved in carbohydrate, purine, pyrimidine, and amino acid metabolism. The mechanisms and signals that control the expression of these genes will also be studied. (1565)

Sponsors: Oklahoma Agricultural Experiment Station, Oklahoma State Regents for Higher Education

PI: Richard Essenberg

Virus Evolution

We test hypotheses about how viruses spread and decline and how new viruses emerge. To achieve this, we analyze the evolution of viral sequences integrated in chromosomes; optimize conditions for detecting multiple viruses in one assay and design and manage probes for such assays; characterize plant viruses by nucleotide sequences; and explore the functions of conserved viral nucleotide sequences. (1789)

Sponsors: Oklahoma Agricultural Experiment Station, USDA-ARS, National Science Foundation, OSU Foundation, IBIS Therapeutics

PI: Ulrich Melcher

Structure/Function and Reaction Mechanism of Bioenergetic Apparatuses

Multiple approaches have been used to study the structure, function, and mechanism of quinone-mediated electron and proton transfer complexes of mitochondrial and photosynthesis electron transfer chains. Significant progresses have been made on the atomic structure of the mitochondrial cytochrome bc1 complex. The structural information obtained has been further confirmed by the study of the site-directed mutagenesis, fast kinetic measurement of electron transfer between the two neighboring components, and other biophysical methods. (1819)

Sponsors: National Institutes of Health, American Heart Association, Dupont Company, Sarkey's Foundation, Oklahoma Agricultural Experiment Station, Oklahoma State Regents for Higher Education

PIs: Chang-An Yu, Linda Yu

Role of Heat Shock Protein 90 in Regulating Protein Kinases

This study will determine the mechanism by which heat shock protein (hsp) 90 and its co-chaperones mediate the maturation of protein kinases. It will also determine whether maturational process can serve as a pharmacological target and whether the maturational pathways for protein folding of these kinases overlap pathways regulation. Identification of novel co-chaperone partners and client proteins will also be made. (1975)

Sponsors: American Heart Association, Oklahoma Agricultural Experiment Station

PI: Robert Matts

The Structure of Pectins from Cotton Cell Walls

This study will include a complete structural analysis of the rhamnogalacturonan region of cotton cell wall pectin. The study will determine how the various subsections of pectins associate with each other and will characterize crosslinks between pectin and xyloglucan. The study will also characterize crosslinks between pectin and the cell wall protein extensin. (2099)

Sponsors: U.S. Department of Energy, Oklahoma Agricultural Experiment Station

PI: Andrew Mort

Improving the Industrial Uses of Oklahoma Wheat

The long-term goal of this research is to understand the basic properties of gliadin (high and low molecular weight glutenin subunits that form the wheat gluten and are directly related to the performance of wheat flour in yeasted baked products). Specific goals include the understanding of the performance of gliadin (high and low molecular weight glutenin subunits in different products), characterization of the interaction of specific groups or subunits with carbohydrates, and the possible correlation of proteins synthesized during the grain filling period to the synthesis of prolamins in wheat. (2351)

Sponsor: Oklahoma Agricultural Experiment Station

Light Regulated Translation and mRNA Stability in Plants

The ability to recognize and respond to extracellular cues is essential for all living organisms
. In plants, one of the most important cues is light, which regulates growth and development through transcriptional and post-transcriptional regulation of nuclear and chloroplast gene expression. One of the best examples of post-transcriptional regulation of a nuclear-encoded mRNA is that of the pea Fed-1 gene, which encodes the chloroplast ferredoxin protein. Fed-1 is regulated by light at the levels of mRNA stability and translation. For Fed-1, light-regulated mRNA stability and translation appear to be conferred by two separate elements. The first element, necessary for destabilization of the mRNA in the dark, is within a region of the Fed-1 5' UTR that contains a (CATT)4 repeat. The (CATT)4 repeat and the surrounding region will be mutated to delineate the mRNA instability element, to test if the function of the instability element is position dependent, and to determine if the element is sufficient to destabilize non-light-regulated mRNAs. To identify proteins involved in regulated Fed-1 mRNA degradation, the instability element will be used in UV-crosslinking assays. The translational control element will be delimited by mutagenesis and, once characterized, the element will be added to non-light regulated mRNAs to determine the minimal sequence sufficient to repress translation in response to darkness. To determine if the translational control element is an mRNA localization sequence, Fed-1 mRNAs that are wild-type or mutant for translational regulation by light will be localized using a GFP system adapted to plants. (2427)

Sponsor: Oklahoma Agricultural Experiment Station

Photosynthetic Electron Transfer Complexes

The overall objective of this research is to elucidate the role of the supernumeral subunit, subunit IV (Mr = 14,384), in the cytochrome bc1 complex from a photosynthetic bacterium Rhodobacter sphaeroides. The specific aims of this project are: 1) to identify amino acid residues of subunit IV involved in interaction with the three-subunit core complex; 2) to identify amino acid residues of cytochrome b involved in interaction with subunit IV; 3) to investigate the effect of subunit IV on the protein conformation and thermostability of the cytochrome bc1 complex by circular dichroism and differential scanning calorimetry; 4) to investigate the protein:lipid interactions in three- and four-subunit complexes; 5) to examine the Q sequestering role of subunit IV using synthetic Q-derivatives; 6) to explore the possible association of supernumeral subunit function with extra segments in the bacterial core subunits; and 7) to compare the 3-D structures of three- and four-subunit bc1 complexes. (2372)

Coupled Enzyme Kinetic Mechanisms and Their Metabolic Consequences

We are studying the kinetic, association, and structural properties of dehydrogenase enzymes. The major objective this year is to elucidate the mechanism for NADH transfer between donor and acceptor pairs of dehydrogenases. In the classical mechanism, the acceptor enzyme can utilize only free NADH (i.e., NADH that is not bound to the donor enzyme). However, a possible alternative is channeled NADH, whereby NADH is transferred directly from donor enzyme to the acceptor enzyme without release of NADH into the bulk medium. The criterion for channeling from the channeling test is R = experimental reaction velocity/velocity predicted assuming the classical mechanism only. We have found that the enzymes tested (glyceraldehye-3-phosphate dehydrogenase, lactate dehydrogenase, alcohol dehydrogenase, alpha glycerol-3-phosphate dehydrogenase) do not channel NADH in contrast to previous appearances. The previous appearances resulted from: 1) side reactions that are present at the very high enzyme concentrations used, and 2) a light-scattering artifact, unavoidably interfering with the 340nm-signal in presence of high protein concentration. The most likely results, when artifacts are avoided, are R values not significantly higher than 1 or 2 (i.e., no channeling within probable experimental errors). (1393)

Sponsor: National Science Foundation, Oklahoma Agricultural Experiment Station

PI: H. Olin Spivey

psycho bitch-nadas

Nadas Moksha
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Post by Nadas Moksha » Sun Nov 05, 2006 5:11 am

FYI 101 #2


Dr Christie Marrian, NRL
DARPA was established in 1958. It is an organization of 240 personnel (approximately 140 of which are technical) and a directly managing a budget of $2 billion. Programmemanagers are generally on a three year secondment. Dr Marrian joined in 1998, and was due to finish his secondment shortly after our visit. In his previous existence he ran the
nanoelectronics facility at NRL, working under Fabian Reese at Stamford on nanoimprinting, and including applications in the patterning of organic materials. He has also been involved in a molecular electronics programme, researching diodes, switches and resettable switches based on organics. DARPA has a policy of encouraging proposals from small businesses, but in this case, nanotechnology is not really a small business activity. DARPA also provides funding for academics, Masters students etc, as feedstock for the economy. It is especially keen on
outcomes-oriented research. Regarding intellectual property, the only proviso is that the Government is given royalty-free licences for its own use.
DARPA is running 5 or 6 programmes related to nanotechnology including:

Materials – for the generation of new properties

Quantum Information for Society and Technology

Advanced Microelectronics

Advanced Lithography


Bionanosesnors for pathogens.

DARPA needs to identify every possible pathogen that an enemy might produce, and be able to counteract it. Projects under Dr Marrian’s supervision include the Molecular electronics programme. With
regard to the procedures for applying for funding, announcements regarding programmes are available on the DARPA website including preliminary call details, full details, deadlines for submission of proposals, then evaluation. Specific deliverables are always identified.
Projects must meet the requirements of ‘domination’ and ‘economic’ – plus also nano! Compare this with the approach of the NSF who are into ‘insight and knowledge’. NSF funding is also smaller in scope, with lower levels of funding. Knowledge and discovery is their prime motivation.
DARPA on the other hand addresses broad projects in selected areas; and funds work aimed at developing ways to solve a given problem

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King Cobra
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Post by RANDY » Sun Nov 05, 2006 6:00 am

Marilyn Tavenner
Secretary of Health and Human Services
Commonwealth of Virginia
1111 East Broad Street
Richmond, Virginia 23219


Ms Marilyn: Tavenner:

Your letter to me dated November 2, 2006, stated that you decided to let the CDC do the job of discovering the cause of “Morgellon’s Syndrome” or “Fiber Disease.” We, the people of Virginia want to do the job ourselves and not to be at the mercy of the CDC and their possible bias. This is our right as Virginians.

The attached letter is by a doctor working for the University that the CDC first asked to conduct their state study of our syndrome. You can see that they are clearly biased. Please ask yourself why the CDC would pick this University to do their study?

The CDC has still been unable to come up with a model, a place for people to go, asked anyone that we know to be studied, developed a hotline for doctors or patients to call that can refer them to physicians aware that this disease is being investigated or to locate doctors that honestly care. We need to know the reason why this is so. This is not acceptable behavior.

We require that a notice be sent out to all health care professionals, especially physicians, alerting them that the CDC is investigating this disease. If these physicians have someone reporting “Morgellon type” symptoms, and have come to an impasse which requires them to suggest psychiatric care causing the patient to leave the office, often in tears, and in contemplation of suicide, that these physicians be aware that there is an investigation in place, a state reporting hotline where these people can register to be part of a study and state database.(434-980-2757)

The CDC is only conducting their study in California. We require an accurate accounting system of those that think they have this syndrome in the Commonwealth of Virginia. The state of Georgia has led the way and I have included the documents they have distributed. There is no excuse why Virginia should not be on board with this program and stop passing the buck to other agencies. It is time for personal and state responsibility.

The numbers of calls I get increase weekly along with suicide attempts due to the horrible lesions, loss of employment and loss of self esteem. Do you not care about these people?

How disorganized is the Commonwealth of Virginia and the CDC? We are not prepared for bioterrorism in the Commonwealth and this syndrome and the lack of attention for its discovery proves this point beyond a showdown of any doubt.

We, the people of Virginia, want for you to do as Georgia has done and alert all physicians state wide and let them know that if they have someone who presents themselves as having this condition (Fiber Disease/Morgellons) that they can contact our state contact so that we can evaluate them. (434-980-2757) They will be referred to a licensed medical professional for evaluation based upon our criteria. Their participation is purely voluntary and not state mandated.

We have a doctor that can do a psych evaluation, a doctor to order the tests, a trained professional to administer a detailed questionnaire and an intake specialist to take photos and document the similarities of the lesions. Labs will be taken and data collected. Results will become part of a nationwide database compiled with other states following our model. We must have an accurate accounting of all of those that feel they are suffering with this “syndrome.”

Can you tell me why this simple model, I have just presented, has not already been created and implemented by the CDC? There is no excuse for this. How many signatures do you want on a petition to ask you to do this? Why should we have to get a petition since this is a no-brainer. Georgia is already doing this. Why not Virginia?

At this point in time all patients in Virginia, reporting this disease, are being force to go onto anti-psychotic medications in order to continue receiving any degree of treatment by a dermatologist. This “pushing” of anti-psychotic medications is a questionable protocol coming from dermatologists and not psychiatrists. These patients are not given any anti fungal, anti parasitic or antibiotics. (I have proof of this claim) The protocol for non-healing skin lesions filled with microscopic fibers begins and ends with anti-psychotic medications. Biopsies are rarely utilized as part of the diagnostic and one sufferer who finally convinced his dermatologist to take a biopsy found skin cancer. With this DOP diagnosis all hope for future treatment is then tainted by this initial incorrect diagnosis. This is horrendous treatment. It is neglect and abuse.

This is not acceptable. We demand better treatment as a group. It is your responsibility to see that this request is carried out. What are you going to do about it? Please do not pass the buck once again.

Randy Yaskal
During the End Times, Good will battle Evil. Where do you stand?

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Post by London » Sun Nov 05, 2006 6:02 am

Wow Nadas!!!! Thank you for your information. I saved them to my computer and will read them again in the morning. So Nadas, what is your take on this illness? Do you have an opinion on the Garlic? ( I posted an anti garlic link yesterday) and was curious of your opinion.

I don't want to be a transhuman, I don't want to extend my life nor do I want it to be maimed from the communications industry either.

I know we have 2 opposing sides here......the wackos aka transhuminist
and the gov't. The gov't has no control over privately funded agendas.

OR>>>>>>do they? You know, the more I think about it, the more I believe it's the gov't blaming them but paying them to do so.

Okay, so here is the fibre channel:

Despite its name, Fibre Channel signaling can run on both twisted-pair copper wire and fiber optic cables.

Fibre Channel Protocol (FCP) is the interface protocol of SCSI on the Fibre Channel.

Fibre Channel layers
Fibre Channel is a layered protocol. It consists of 5 layers, namely:

FC0 The physical layer, which includes cables, fiber optics, connectors, pinouts etc.
FC1 The data link layer, which implements the 8b/10b encoding and decoding of signals.
FC2 The network layer, defined by the FC-PI-2 standard, consists of the core of Fibre Channel, and defines the main protocols.
FC3 The common services layer, a thin layer that could eventually implement functions like encryption or RAID.
FC4 The Protocol Mapping layer. Layer in which other protocols, such as SCSI, are encapsulated into an information unit for delivery to FC2.
FC0, FC1, and FC2 are also known as FC-PH, the physical layers of fibre channel.

Fibre Channel products are available at 1 Gbit/s, 2 Gbit/s and 4 Gbit/s. An 8 Gbit/s standard is being developed. A 10 Gbit/s standard has been ratified, but is currently only used to interconnect switches. No 10 Gbit/s initiator or target products are available yet based on that standard. Products based on the 1, 2, 4 and 8 Gbit/s standards should be interoperable, and backward compatible; the 10 Gbit/s standard, however, will not be backward compatible with any of the slower speed devices.

Fibre Channel Infrastructure
Fibre Channel switches are divided into two classes of switches. These classes are not part of the standard, and the classification of every switch is left up to the manufacturer.

Director switches are characterized by offering a high port-count in a modular (slot-based) chassis with no single point of failure (high availability).
Fabric switches are typically fixed-configuration (sometimes semi-modular) non-redundant switches.
Brocade, *Cisco and *McData provide both Director and fabric switches. *QLogic provides fabric switches. If multiple switch vendors are used in the same fabric, the fabric will default to "interoperability mode" where some proprietary advanced features may be disabled.

[edit] Fibre Channel Host Bus Adapters
Fibre Channel HBAs are available for all major open systems, computer architectures, and buses, including PCI and SBus (obsolete today). Each HBA has a unique World Wide Name (WWN), which is similar to an Ethernet MAC address in that it uses an Organizationally Unique Identifier (OUI) assigned by the IEEE. However, WWNs are longer (8 bytes). There are two types of WWNs on a HBA; a node WWN, which is shared by all ports on a host bus adapter, and a port WWN, which is unique to each port. Some Fibre Channel HBA manufacturers are Emulex, LSI Logic, QLogic and ATTO Technology ATTO was the first to market with 4Gb and Quad Channel Host Adapters.

There is more but that's a start.....

AND THIS IS THE cDc I said, my friend went to college with the founder. He now lives in Harlem.

CULT OF THE DEAD COW, also known as cDc, is a computer hacker and DIY media organization founded in 1984 in Lubbock, Texas. The group maintains a weblog on its site, also titled "CULT OF THE DEAD COW." New media are released first through the blog, which also features thoughts and opinions of the group's members.

To further the Cult's stated goal of "Global Domination Through Media Saturation," over the years cDc members have granted interviews to major newspapers, print magazines, online news sites, and international television news programs.

*****NOTE: This group claims responisibility for Reagan getting Alzheimers via a blowgun.

So, Randy who are you with? Not much of a choice is there?


Look at this. Does this look familiar to you? Please let me know. It soes to me. ... 0/mico.JPG
Last edited by London on Sun Nov 05, 2006 6:08 am, edited 1 time in total.

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King Cobra
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Post by RANDY » Sun Nov 05, 2006 6:05 am

That has nothing to do with what we have.

Test for it. Prove it. It is just a lot of blah blah. Not connected to us at all.

You made it all up in your head. Connected it without any medical scientific tested proof.
During the End Times, Good will battle Evil. Where do you stand?

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Post by London » Sun Nov 05, 2006 6:58 am

WEll, Well, Well.........Looks like I was right. I;'m referring to the part where I stated way back at the end of May that this does indeed have to do with the wasp and honey bee......and,,,,,,,the wheat and barley!!!

Sure does.....just checked my work again tonight. In fact if you don't think I know what in the hell I'm talking about, go back up to my last hyperlink where it opens up to a picture......

Now does that not look familiar??????????????? Can anyone name the hair looking thing in the photo.

This is all about the wheat and b arley. Why they thru the silicate and computer bullshit in with it and the solar fusion, I don;t know.

People, you got a serious plant disease on your hands.



LYCTIC ENZYMES!!!!!!!!!!!!!!!!!!!!!!!!!!!!!!!!!!!! WAIT AND SEE..........WAIT AND FRIGGIN SEE!!!!!!!!!!!!!!!!!!!!!!!!!!!!!!


Nadas Moksha
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Post by Nadas Moksha » Sun Nov 05, 2006 7:48 am

london i think the web that binds is doped films layered with programmable enzymes that form our "chips" ... . when ion interacts with genetic spin pattern coupled metabolic rate is what renders a taylor made electronic silicomorph stealth pathogen to suite.. . . . . . .this could even manufactor your wasp from random simple sugars and carbs the missed the trash bin... . . or just reanimate unused parts from deceased plants/insecta for hybred delivery in case of self inflicted famin
sratigy... ..
Regarding the potential environmental impacts of GMO, it is necessary to understand the biology of the GMO as well as its relationship with the environment where it will be released. The information on the basic biology of the plant and their interactions with the environment including genetic characteristics, reproductive biology, the center of origin and biodiversity, pests, diseases, and ecological characteristics are important issues in evaluating environment safety. All these issues must be taken into account in the evaluation of the GMO in relation with the potential risks to the environment.
2002: Australia - 15 kgs of Monsanto's GM cotton seed was spilled
during transport Monsanto's GM Bollguard cotton seed had spilled into a trailer which was then cleaned out and the seed thrown into a rubbish bin by an employee who was unaware that it was GM cotton.


Drug delivery platforms based on nano-sized geometries (aka nanovectors) promise a revolutionary impact on the diagnosis and therapy of various types of cancers and diseases. To date, polymeric nanovectors (e.g., nanoparticles, nanospheres, nanocapsules,
nanosuspensions, micelles, liposomes, and dendrimers) have been the focus of intense research. Since the mid-19th century, inorganic layered double hydroxide (LDH) ceramics are used for a variety of applications including antacids, catalysts, anion exchangers, and adsorbents. The intercalated anion, within the interlayer space between the cation hydroxide layers, can be readily exchanged with a variety of negatively charged bio-molecular agents such as carboxylic acids, amino acids, nucleoside phosphates, proteins, DNA, ATP, vitamins,
and drugs. Therefore, since 2001, bio-hybridized LDH nanoparticles (or LDHNs) have been evaluated as a passive vector (administrable via the intravascular or pulmonary route) fo rdrug and non-viral gene delivery, but its development is at a stage of infancy. To be
competitive with alternate platforms, research on LDHN-based nanovectors must focus on safety, active targetability, and efficacy. Moreover, it is imperative to build in multimodality with respect to therapy and imaging, while maintaining overall structural simplicity and
economics of synthesis. This presentation will first specify the advantages of LDHN as a nanovector for passive targeting, as well as issues in and attributes of LDHN for active targeting. The judicious selection and synthesis of physiologically relevant LDHN compositions will be discussed. Next, the surface activation of LDHN, a critical first step towards surface functionalization for active targeting, will be illustrated. Finally, a few
strategies to build in multimodalities will be described and preliminary experimental results will be presented.

GM Contamination Register Report 2005

There are 17 illegal releases included in the register which are associated with research and development
or black-market growing (in India, Brazil and Romania). Mistakes and errors in handling are one
apparently common cause of illegal releases associated with research and development. Failures in
inspection and enforcement of controls on field trials have also been highlighted in a 2005 USDA review
of its own systems.
Eight reported and verified cases of adverse agricultural side-effects have been reported with GM crops,
affecting the USA, Argentina, Canada and Australia. These include the emergence of herbicide-tolerant
weeds in the USA and Argentina, unreliable performance of Bt cotton in India, and the first field case in
Australia of cotton bollworm resistance to a toxin, Cry1Ac, used in GM cotton.
The data from the GM Contamination Register show that GM contamination can arise at every stage of
development – from the laboratory, to the field, to the plate. Cases of misidentification, poor quality
control and lack of awareness of proper controls in laboratories have led to GM tomato, zucchini and
maize seed being distributed around the world and meat from GM pigs entering the food chain. Seed used
for GM field trials, even the high-profile scientific farm-scale evaluations in the UK, has been found to be
contaminated by other GM crops. Experimental trials have led to contamination of neighbouring and
subsequent crops. Cross-pollination and poor quality control have led to non-GM seed and food aid being
contaminated. Illegal large-scale growing of GM crops in Brazil, India and Romania, together with
scientists conducting illegal trials or failing to contain them properly, show that GM organisms are often
out-of-control even when claimed to be ‘strictly contained’.
The Bt10 maize contamination incident in 2005 reveals a particular problem with detection and
prevention of GM contamination. In official terms, this GM maize did not exist. It had not been tested in
field trials, so no details had to be disclosed to authorities to gain authorisation. Even if it had been used in
trials, it is unlikely that information about the construct and genes inserted would have been in the public
domain, as this is often deemed ‘confidential business information’. This has become standard practice
only over the past years. At the same time an increasing array of potentially dangerous genes with respect
to human health are being introduced into crops – coding for drugs or other biologically active compounds that could easily escape detection. Poor controls of trials with such GM drug producing crops were also highlighted by the USDA.

US shipping unwanted GE grains as "Food Aid" to Latin America. Organic
Consumers’ Association, June 2001 ... 112801.cfm
2002: Bolivia - StarLink maize - a GM maize intended for animal
feed was found in US food aid.
Sampling of US food aid sent to Bolivia found StarLink GM
maizecontamination at levels around the limit of detection - 0.1%.
StarLink maize was grown in the USA for animal feed but was also
found in food products. The StarLink maize, produced by Aventis (now
Bayer), is genetically modified to contain a gene from the bacterium,
Bacillus thuringiensis, coding for an insecticidal Bt toxin known as
Cry9C. This particular type of Bt toxin is not found in other GM insect
resistant crops and there are concerns that it could be a human allergen
because it is heat stable and does not break down in gastric acid in the
human digestive system - characteristics shared by many allergens
The StarLink Situation. Iowa Grain Quality Initiative 18 November 2003 ... arlink.htm


King Cobra
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Post by London » Sun Nov 05, 2006 7:55 am

oh Thank you, thank you, thank you......I do so appreciate that thought.
And Nad, althought I never posted about it, I did witness small yellow jacket wasp coming grom my computer. I swear if I'm lying I'm dying.
This was back in June or July!

Also saw your write up on Maize and Australia. I thought we imported bad corn to them. Hey check this out.....

here is mayb e why they were pushing the ivermenctin:

Characterization of a Chitinase Gene from Stenotrophomonas maltophilia Strain 34S1 and Its Involvement in Biological Control

Received 19 July 2001/ Accepted 17 December 2001

A chitinase gene was cloned on a 2.8-kb DNA fragment from Stenotrophomonas maltophilia strain 34S1 by heterologous expression in Burkholderia cepacia. Sequence analysis of this fragment identified an open reading frame encoding a deduced protein of 700 amino acids. Removal of the signal peptide sequence resulted in a predicted protein that was 68 kDa in size. Analysis of the sequence indicated that the chitinase contained a catalytic domain belonging to family 18 of glycosyl hydrolases. Three putative binding domains, a chitin binding domain, a novel polycystic kidney disease (PKD) domain, and a fibronectin type III domain, were also identified within the sequence. Pairwise comparisons of each domain to the most closely related sequences found in database searches clearly demonstrated variation in gene sources and the species from which related sequences originated. A 51-kDa protein with chitinolytic activity was purified from culture filtrates of S. maltophilia strain 34S1 by hydrophobic interaction chromatography. Although the protein was significantly smaller than the size predicted from the sequence, the N-terminal sequence verified that the first 15 amino acids were identical to the deduced sequence of the mature protein encoded by chiA. Marker exchange mutagenesis of chiA resulted in mutant strain C5, which was devoid of chitinolytic activity and lacked the 51-kDa protein in culture filtrates. Strain C5 was also reduced in the ability to suppress summer patch disease on Kentucky bluegrass, supporting a role for the enzyme in the biocontrol activity of S. maltophilia.

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King Cobra
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Post by RANDY » Sun Nov 05, 2006 8:04 am

Treatment and Control

Antibiotic sensitivity of the causative organism of Tyzzer's Disease has been highly variable but tetracycline drugs appear to be an effective means of treatment.

A very simple test to take to define and a simple cure.
During the End Times, Good will battle Evil. Where do you stand?

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King Cobra
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Joined: Sat Jan 21, 2006 8:16 am

Post by RANDY » Sun Nov 05, 2006 8:12 am,1607,7-15 ... --,00.html

Nothing on this thread has ANTHING to do with our disease. this is because no one on here has any background in science and you have no clue to what you are reading and all of it is DISinformation.

Readers beware......dis-information defined: ... tion.shtml

This has a simple solution without a TAM AUTHORITY or too complex for anyone to grasp.

Take Tam and compare him to all these tactics (knows it all/expert/ but will not tell thewhole truth) and then take Londons outrage if anyone goes against her and Nokas ramblings and you will see what a great team they truly are people. WAKE UP! They have this entire page down pat!

During the End Times, Good will battle Evil. Where do you stand?


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