The Fiber Disease

Human Anatomy, Physiology, and Medicine. Anything human!

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Post by Sabrina » Tue Oct 24, 2006 4:28 pm

Thanks for the info Nettimo. Never heard of her but with a title like that it could only be a real flop. :D

I was inspired today so I wrote this.

[email protected]

Dear New York Times,

I have always admired your professionalism and very much appreciate the invaluable service that you provide to the public. Thank you!

Today however, I felt compelled to write to you to point out some very major, detrimental, and punitive errors in an article published here: ... ref=slogin

Is It Disease or Delusion? U.S. Takes on a Dilemma
Published: October 24, 2006

“Earlier this year, a young man in Texas reportedly committed suicide after struggling with what his mother has described as Morgellons.”

I am wondering where your reporter got this information because this is not true at all. Perhaps they should have investigated and contacted his Mother to asked her what really happened to her son Travis.

The truth is we don’t know what killed Travis but suicide was a premature assumption and has defiantly been ruled out and long ago. The truth is we do not know if this disease kills or not because no one has investigated or done any studies on this yet and if they have then they sure are not telling.

I will agree that suicide sounds better than the fact that we have a serious epidemic spreading all across America and other countries that we know almost nothing about and the CDC still cannot get a handle on. But suicide, no, NOT TRUE, this is just not the case here.
Please amend your article accordingly.

One other extremely important point is that your reporter wrote about a delusional disorder when addressing this clinical condition. Infected individuals are able to expel fibers (red, white, blue, and black) from their skin that everyone is able to witness. Many doctors have witnessed this first hand as well. I invite you to look for your self just as Brandi Koch offered,”the show of a life time.” Why not take her up on it?

For that matter, I am sure any infected individual would be happy to show you how this manifests through the skin. Do you not agree that your reporter should have asked to see this for himself so he could report this story like a professional journalist?

Now there is a concept, how about doing some investigating and witnessing this phenomena first hand so that this extremely important issue can be reported properly!

Please inform your reporter that delusions cannot be seen, touched, observed or video taped and that psychosomatic conditions cannot produce solid matter such as this either. (where did you find this guy?)

One question I have is if your reporter did actually investigate this and see it for him self, would he be considered delusional also? Perhaps this is the reason this crucial, basic, and common sense step to professional investigative reporting was obviously left out of this story. This simple task would have certainly ruled out the unfounded accusations of being delusional. Then your agency would really have a huge story and an accurate one too.

Many people who have this condition would be willing to submit to any interviews and/or observations that you may request in order to rule out that the growing mass of sufferers are not mentally ill and definitely not imagining this medical phenomenon.

We have a very real and bizarre epidemic on our hands. When will you allow the truth to be told?

Please feel free to contact me if you have any further questions and please amend the article as I have requested.

Sabrina ~Fiber Disease Victim

I invite you all to write to them as well, let them know how you feel about this injustice.


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Post by Skytroll » Tue Oct 24, 2006 6:21 pm


My gosh.....what the heck is going on? Watch your back there. Sounds like the nannies want to control all the babies........If not by fear by other means. This is nasty.....

For all of you here is a No Fear Act you might be interested in........


Found more after reading article on P. Aeruginosa

This talks of hydrogen cyanide biosynthetic gene.
and Catabolism.

So found this:
"Pseudomonas aeruginosa is a gram-negative bacterium that can cause serious infections in patients suffering from cystic fibrosis, cancer, infection with human immunodeficiency virus, or severe burn wounds (16, 29). The pathogenesis of P. aeruginosa infections is due to the production of both cell-associated and extracellular virulence factors. One of these extracellular compounds, hydrogen cyanide (HCN), has been found at relatively high concentrations in patients with freshly infected burns (27). Evidence for HCN being a virulence factor comes from an experimental infection model in which an hcn insertion mutant of P. aeruginosa had a strongly reduced ability to kill the nematode Caenorhabditis elegans (L. Gallagher and C. Manoil, Abstr. Pseudomonas '99: Biotechnol. Pathog., abstr. 75, 1999). Cyanide is a potent inhibitor of cytochrome c oxidase, the terminal component of the aerobic respiratory chain in many organisms, and of several other important metalloenzymes (52)."

Long article but this above tells us that a cyanide is the cause of the breakdown inside our bodies.

Catabolism is the metabolic process that breaks down molecules into smaller units. It is made up of degradative chemical reactions in the living cell. Large polymeric molecules (polysaccharides, nucleic acids and proteins) are processed into their constituent monomeric units (i.e. monosaccharides, nucleotides and amino acids, respectively).

Cells use monomers to construct new polymeric molecules and disassemble them to simple cellular metabolites (lactic acid, acetic acid, carbon dioxide, ammonia, urea, etc.). The creation of cellular metabolites is an oxidation process involving a release of chemical free energy, not all of which is lost as heat, but some of which is partially conserved through the coupled synthesis of adenosine triphosphate. The hydrolysis of this compound is subsequently used to drive almost every energy-requiring reaction in the cell. Catabolism provides the chemical energy necessary for the maintenance of the living cell. Examples of catabolic processes include breakdown of muscle protein in order to use amino acids as substrates for gluconeogenesis and breakdown of fat in adipose to fatty acids.

Because it is counterproductive to have anabolic and catabolic processes occurring in cells simultaneously, there are many signals that switch on anabolic processes while switching off catabolic processes and vice versa. Most of the known signals are hormones and the molecules involved in metabolism itself. Endocrinologists have traditionally classified many of the hormones as anabolic or catabolic."

more definitions:


"The secondary metabolite hydrogen cyanide (HCN) is produced by Pseudomonas fluorescens from glycine, essentially under microaerophilic conditions. The genetic basis of HCN synthesis in P. fluorescens CHA0 was investigated. The contiguous structural genes hcnABC encoding HCN synthase were expressed from the T7 promoter in Escherichia coli, resulting in HCN production in this bacterium"

"HCN synthase were expressed from the T7 promoter in E. coli"

So, if we can find more on this angle then the rest should follow. This adapts in the environment.

Watching the video, I see the total connectiont to protists, in particular the "little animacules"

More on that later,


Hang tight there, and know we are here.

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Post by London » Wed Oct 25, 2006 1:35 am

Sorry to offend anyone. I meant every word I said in two of those post from me, but that third one? I do not even remember! Sorry once again, espeically to snowflake.

Look, I think these are Tams old C3 firbres



Discharge patterns of human C-fibers induced by itching and burning stimuli

H. O. Handwerker, C. Forster and C. Kirchhoff
Institut fur Physiologie und Biokybernetik, Erlangen, Federal Republic of Germany.

1. The aim of this investigation was to study the peripheral neural mechanisms of the C-fiber-mediated modalities of burning pain and itch by the use of microneurography of human unmyelinated afferents. 2. Sixteen stable recordings of single C-fibers and 6 multiunit recordings were obtained from the superficial radial nerves of volunteers. All units were excited by stimulating their receptive fields with von Frey bristles (range 10-600 mN), and all but four units were also driven by radiant heat stimulation. 3. Histamine was iontophoretically applied to the receptive fields of these units for 20 or 30 s and was found to provoke itching sensations lasting several minutes, together with wheal and flare responses. Subsequently a solution containing 20 or 30% mustard oil was applied to the receptive field of the respective unit, which provoked a sensation of burning pain. 4. One-half of the units were excited by histamine, and the median discharge rates derived from interspike intervals ranged from approximately 0.1 to 0.8 Hz. Mustard oil-induced activity was observed in all histamine-sensitive units and also in three single units and in one multiunit recording that revealed no histamine response. Median interval-derived discharge rates ranged from 0.2 to 1.2 Hz. 5. Analysis of the interspike interval distribution and of the autocorrelation function derived from the chemically induced discharges of single units provided no evidence for an encoding of itch and burning pain in different discharge patterns of units responding to histamine and to mustard oil.(ABSTRACT TRUNCATED AT 250 WORDS)

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Post by Skytroll » Wed Oct 25, 2006 3:14 am


Okay so what is a C-fiber?

C fiber
Any of the unmyelinated fibers, 0.4 to 1.2 micrometers in diameter, conducting nerve impulses at a velocity of 0.7 to 2.3 meters per second.

okay, 7 to 2.3 m/sec

Now, if those are myelinated:
myelin sheath
The insulating envelope of myelin that surrounds the core of a nerve fiber or axon and facilitates the transmission of nerve impulses. In the peripheral nervous system, the sheath is formed from the cell membrane of the Schwann cell and, in the central nervous system, from oligodendrocytes. Also called medullary sheath."

Okay, now.......if we go a little further......

Microneurography (Hagbarth, Vallbo): Microneurography is a method to record the traffic of impulses in human nerves with percutaneously inserted needle electrodes. The method was developed by Hagbarth and Vallbo in the late 60s in the department. They were able to record single units from individual axons in human nerves with electrodes manufactured from tungsten wires. They did the pioneer work of developing the method recording from muscle afferents such as muscle spindles. This project continued to develop over the years and recruited new collaborators from different parts of the world. The microneurographic method has been used widely internationally and has given insight into a number of questions regarding muscle spindle function in normal and in patients with spasticity or rigidity.
The method has also been used to explore a number of neural systems in healthy human volunteers, e.g. proprioceptive, tactile, nociceptive and autonomic mechanisms" ... rch/micron

A nociceptor is a sensory receptor that sends signals that cause the perception of pain in response to potentially damaging stimulus. Nociceptors are the nerve endings responsible for nociception, one of the two types of persistent pain (the other, neuropathic pain, occurs when nerves in the central or peripheral nervous system are damaged). When they are activated, nociceptors can trigger a reflex. LocationNociceptors are sensory neurons that are found in external tissues such as skin, cornea and mucosa as well as in internal organs, such as the muscle, joint, bladder and gut. The cell bodies of these neurons are located in either the dorsal root ganglia or the trigeminal ganglia. Types and functionsThere are several types of nociceptor and they are classified according to the stimulus modalities to which they respond: i.e. thermal, mechanical or chemical. C fiber axons. Thus, pain often comes in two phases, the first mediated by the fast-conducting Aδ fibers and the second part due to C fibers. Silent nociceptors do not usually fire action potentials, though they are much more likely to do so in the presence of inflammation or damaging chemicals (Kandel et al, 2000). Together these nociceptors allow the organism to feel pain in response to damaging pressure, excessive heat, excessive cold and a range of chemicals, the majority of which are damaging to the tissue surrounding the nociceptor. PathwayAfferent nociceptive fibers (those that send information to, rather than from the brain) travel back to the spinal cord where they form synapses in its dorsal horn. The cells in the dorsal horn are divided into physiologically distinct layers called laminae. Different fiber types form synapses in different layers. Aδ fibers form synapses in laminae I and V, C fibers connect with neurons in lamina II, Aβ fibers connect with lamina IV. Information is then sent from the spinal cord to the thalamus and the cerebral cortex in the brain. ReferencesKandel E.R., Schwartz, J.H., Jessell, T.M. 2000. Principles of Neural Science, 4th ed., pp.472-479. McGraw-Hill, New York.
Nervous system - Sensory system - Somatosensory system - [ edit]
Spinal pathway: Somatosensory information
Medial lemniscus: Touch (Pressure & Vibration) | Proprioception
Spinothalamic tract: Pain | Temperature
Touch: Pacinian corpuscles | Meissner's corpuscles | Merkel's discs | Ruffini endings | Free nerve endings | Hair follicle receptors
Proprioception: Golgi organ | Muscle spindle (Intrafusal muscle fiber)
Pain: Nociceptors Temperature: Thermoreceptors

Just a few definitions.


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Post by Skytroll » Wed Oct 25, 2006 4:28 am

hair follicle receptors:

Stem cells are located at the junction of the arrector and the follicle, and are principally responsible for the ongoing hair production during the Anagen stage.

Certain species of Demodex mites live in the hair follicles of mammals (including those of humans) where they feed on sebum.

[edit] Hair growth phases
Hair grows in cycles of various phases: anagen is the growth phase; catagen is the involuting or regressing phase; and telogen, the resting or quiescent phase. Each phase has several morphologically and histologically distinguishable sub-phases. Prior to the start of cycling is a phase of follicular morphogenesis (formation of the follicle). There is also a shedding phase, or exogen, that is independent of anagen and telogen in which one of several hairs that might arise from a single follicle exits. Normally up to 90% of the hair follicles are in anagen phase while, 10–14% are in telogen and 1–2% in catagen. The cycle's length varies on different parts of the body. For eyebrows, the cycle is completed in around 4 months, while it takes the scalp 3–4 years to finish; this is the reason eyebrow hairs have a fixed length, while hairs on the head seem to have no length limit. Growth cycles are controlled by a chemical signal like epidermal growth factor."

more on hair follicles:

following the sensory:

The proprioceptive sense is believed to be composed of information from sensory neurons located in the inner ear (motion and orientation) and in the stretch receptors of joints and muscles (stance). There are specific nerve receptors for this form of perception, just as there are specific receptors for pressure, light, temperature, sound, and other sensory experiences.

Although it was known that finger kinesthesia relies on skin sensation, recent research has found (Robles-De-La-Torre & Hayward, 2001) that kinesthesia-based haptic perception strongly relies on the forces experienced during touch. This research allows the creation of "virtual", illusory haptic shapes with different perceived qualities (see the MIT Technology Review article The Cutting Edge of Haptics

Now back to the C-fiber. Three levels of epidermis?
picture: ... /9620/FIG1

from this link:

Now I do not know if this is the C-3 but I am beginning to wonder.


Think this pertains, London?
"Shaposhnikov and A. V. Zeveke

Received: 31 January 1975

Abstract Afferent activity in thin myelinated and unmyelinated cutaneous nerve fibers was analyzed by an impulse collision method and by methods improving the signal-to-noise ratio in the record of the antidromic action potential. The following groups were distinguished among the thin myelinated and unmyelinated nerve fibers on the basis of the results of investigation of conduction velocities, thresholds of electrical excitation, and response to mechanical stimulation: A 1 (conduction velocity 30-14 m/sec) — a relatively larger number of these fibers conducts excitation in response to weak mechanical stimulation; A 2 (14–4.0 m/sec) — the receptors of these fibers are more easily excited by a strong stimulus; a group of "mixed" fibers, containing myelinated and unmyelinated nerve fibers (4–2 m/sec), conducting excitation in response to both types of mechanical stimulation; C1 (2.0–1.0 m/sec) — a fairly large number of these unmyelinated fibers conducts impulses in response to weak mechanical stimulation; C2 (1.0–0.15 m/sec) the majority of fibers of this group is connected with receptors requiring strong mechanical stimulation for their excitation.
Research Institute of Applied Mathematics and Cybernetics, N. I. Lobachevskii State University, Gor'kii. Translated from Neirofiziologiya, Vol. 8, No. 1, pp. 67–75, January–February, 1976.

So we just might become electrical bodies for the peruse of who, again?

New book speaking of above:

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Post by Skytroll » Wed Oct 25, 2006 4:44 am

So, In conclusion........

I am ......ur......thinking that if the velocity is increased by some form of electrical stimilus..

from .7 to 2.3 m/ unmyelinated

now consider from the above article:

A 1 (conduction velocity 30-14 m/sec) — a relatively larger number of these fibers conducts excitation in response to weak mechanical stimulation; A 2 (14–4.0 m/sec) — the receptors of these fibers are more easily excited by a strong stimulus; a group of "mixed" fibers, containing myelinated and unmyelinated nerve fibers (4–2 m/sec), conducting excitation in response to both types of mechanical stimulation; C1 (2.0–1.0 m/sec) — a fairly large number of these unmyelinated fibers conducts impulses in response to weak mechanical stimulation; C2 (1.0–0.15 m/sec) the majority of fibers of this group is connected with receptors requiring strong mechanical stimulation for their excitation

So, the higher mechanical stimulation the more stimulated the nerves become. Now, If outside electrical or even consistent low frequency is constantly applied, what does that do to the myelin sheath? Or if there is unmyelinated nerve fibers that increase would most likely destroy them.

Red could be those still alive, black dead ones, blue the markers to register how high the voltage.

Far fetched, but, this is related to the nerve fibers in the skin, the sensory fibers, etc.

Crazy or fumbling?


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Post by RANDY » Wed Oct 25, 2006 5:28 am

Recent discovery from polling people....gonna try polling here.

It appears that those with an RH factor that is positive catch this in households where those without the monkey protein do not.(RH negative)

It does not matter what blood type you have (A-B-AB-O) but the factor may play a role. Those with RH ( the monkey) have a certain protein those with RH negative do not have this protein. This protein may feed this disease.

That is why you see moms and kids with it. Usually they are one factor. Also if the dad is negative and mom is postive it can cause trouble and I am wondering about the PH of those that have lost while carrying.

How many people here have rh positive blood and how many have family members that do not have this, with RH negative blood?

My observation so far is that 100% of those with RH positive blood have caught this or have this and those with RH negative blood are immune. I only have a small sampling of friends and famly to pull from so far.

Feeedback needed. Honest feedback if you KNOW what factor you have and the factor of those who have caught this and those that appear to be immune.


If you do not want to report here, please write me at [email protected]

During the End Times, Good will battle Evil. Where do you stand?

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Post by Sabrina » Wed Oct 25, 2006 5:47 am

Oh Oh Oh!!!! Great things are happening!!!!!!! Doors are opening! :D

Did you guys catch the Rense show? OMG!!!! What potential! New hope for treatment!

We are moving in many direction. THANK YOU EVERYBODY!!!!

Thought I would pass this along to all of you. I recieved it at 3:38 this afternoon.

Dear Ms. Sabrina

Thank you for reading The Times, and for your kind comments. I will alert the editors who handled Mr. Mason's article and make sure they see the points you have raised.

When we run corrections, they appear on Page A2 of the print editions. And the day they are published, you can find them under the "Corrections" label on the home page of If the editors determine that no correction is warranted, someone will be in touch to explain why that is the case.

We depend on readers like you, Ms. Sabrina, to keep us on our collective toes and insist that we uphold the standards you have come to expect of The Times.

Best regards,

Greg Brock
Senior Editor

BCC: Science Desk.

8) 8) 8)

Sky, thanks for the words of encouragement, I needed them.

London, are you still alive? (just kidding… I think) Please check in periodically.

Snowflake, Welcome to biology on line. Forgive my delay in greeting you.

Tamtam, what’s your view on the silicon claiming to be found in the new lab reports?

Randy, never mind, I didn't read your post.


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Post by tamtam » Wed Oct 25, 2006 5:29 pm

Try this type association:

exopolymer matrix, pigmented filament, silica, silicon etc etc.
Its a synthetic micro organism (GMO/ select agent)
No literature is readily available as I hope you do understand.

The netting you see can be associated with a wing matrix.
Check: wing vein(ation)

JSTOR: Silicon Compounds in Biological Systems
B. 171, 19-30 (1968) Printed in Great Britain Silicon compounds in biological systems BY A. C. ALLISON Clinical Research Centre Laboratories, Mill Hill, ... sici?sici=0080-4649(19680813)171%3A1022%3C19%3ASCIBS%3E2.0.CO%3B2-X - Similar pages

Copy extract in google
Last edited by tamtam on Wed Oct 25, 2006 6:42 pm, edited 3 times in total.

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Post by Barz » Wed Oct 25, 2006 5:32 pm

Hi Randy,

Please call your contact, Dan Rutz and ask him why Jan Smith has had to hire her own pathologist to do her own study to find out what is going on with her "dillusions". This is outrageous. Please tell him that his agency is good for nothing and that if we have to put our faith in his system - the one we pay him for, as it is now, we might as well live in Zaire and get better treatment.

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Post by tamtam » Wed Oct 25, 2006 5:37 pm

Identifying Non-Targets From Fall Armyworm Pheromone Captures ...
... worn and tattered, with many of the wing and body scales missing. ... of L. phragmatidicola has a rough texture with wing veination clearly visible. ...

Copy this link in Google and check figure 12

Photo 12: The main "bone" of this wing relates to what is expressed in the mold as a fiber (bone/ vein)
Pigmentation seems to relate most to the signal colors of an insect wing.

The 2D steps on the wing relate to 3D expressions.
Last edited by tamtam on Wed Oct 25, 2006 5:58 pm, edited 4 times in total.

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Post by tamtam » Wed Oct 25, 2006 5:49 pm

Developmental Biology Online: Gradient Models of Positional ...
(A) Photograph of an eyespot on the wing of Morpho peleides. (B) Diagram of a two-gradient model that may explain the way the spot was generated. ...

Gradient Models of Positional Information

How can cells be informed of their position in the embryo and then use that information to differentiate into the appropriate cell type?

Excerpt article:

(Concerns article: 3.3 Gradient Models of Positional Information)

This is the link: ... informatio


Other links/ titels:

A model for colour pattern formation in the butterfly ... - Sekimura
Formation and maintenance of distinctive cell patterns ... - Honda
Development and evolution on the wing - McMillan


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