cell-surface proteins that bind gaba with high affinity and trigger changes that influence the behaviour of cells. Gaba-a receptors control chloride channels formed by the receptor complex itself. They are blocked by bicuculline and usually have modulatory sites sensitive to benzodiazepines and barbiturates. Gaba-b receptors act through g-proteins on several effector systems, are insensitive to bicuculline, and have a high affinity for l-baclofen.
Results from our forum
Can somebody explain me the reason why the inhibitory activity of GABA's receptors is coupled to excitation? Why is there no effect of GABA signal if it is not near ...
See entire post