Human Anatomy, Physiology, and Medicine. Anything human!
or look here:
http://www.google.com/search?q=Human+sk ... f8&oe=utf8
and....here to for photo:
http://images.google.com/images?q=Human ... a=N&tab=wi
My theory is only a theory that I feel in my gut it is true and seems to make the most sense to me.
I know that a company is NY State manufatured a pesticide or herbicide..I don't know if it was to kill the bugs or help the plants. My belief leads me to think to strengthen the plants.
It is just a theory though. Citovsky's agrobacterium theory will be backed by facts if he gets the NIH grant. IF we are now part plant..then maybe I am right..I don't know..wish I did.
That is all I really want...to figure this out in a logical maner and follow through on one theory at a time or many theories at a time but at least follow through on the theories and not jump around. Take it ot the finish line and if you reach a dead end..start again.
I just want to be able to wear short sleeves in the summer and hug my friends and their new babies and go on vacations and be a real person again withut fear of infecting another.
I have had this for 18 years, 10 of remission. The new strain seems to have a more viotile effect on the people it attacks. It is mutating and does mutate to anything we use to remove it. It usually comes back with a vengence.
We need to FOCUS. That is my point. Never was anything different. FOCUS and FOLLOW THROUGH.
During the End Times, Good will battle Evil. Where do you stand?
http://www.google.com/search?q=human+mi ... f8&oe=utf8
Now herel='s one from someone in wisconsin. That be par for the course since it is there that the University created this crap
HUH KEELY???? Hey girl, I hAVE HAD SOMEONE WATCHING YOU FOR NEARLY 2.5 WEEKS NOW.....WHAT COMES AROUND GOES AROUND GIRLFRIEND.
http://www.themanufacturer.com/us/detai ... ts_id=4352
and.....to learn more:
http://www.intermec.com/eprise/main/Int ... /RFID/RFID
is it a BINGO????
http://www.media.mit.edu/physics/public ... merman.pdf
http://www.google.com/search?q=The+safe ... f8&oe=utf8
Love to you....
I'm back after resting from three days of attack on this board and at home. All is in the hands of attorneys now so attack away if you so wish. I want to thank the lovely people of this board for defending me.
I originally came, not only to defend myself but to post some new info. I found on nanotechnology after getting some ideas from Jan's samples after the rense show and their use by certain companies of which I have no problem naming, they advertise as communication devices as well as other uses and are open to the free market. I've got to get cleaned up after resting from my assault. I do believe this person has been duly warned that she is now being monitored by a number of individuals, companies and their legal firms and hopefully will stay within reasonable bounds on this site and others. She has been blocked from my personal email as that was as full of the caca as was this site. I have many abstracts I'd like to share. I want to go back and make sure I'm not duplicating effort but London, Tam, msc and others....I think you will find them very interesting and thus the way they can be introduced and activated, very simplistic. And I didn't see my answer to you London, but I am just waking and scanned quickly but yes, these UNUSUAL sirens, living out in the boondocks, are driving me CRAZY! The seem to sensitize and effect the vibrational sensations and tinnitus.
I'll be back after my cleanse.
Love you all for the work you do!
ps. I'll bring back the company names to google.
And as for united...my definition is moving forward towards a common cause despite differences of opinion and in that moving forward, agreeing that disagreeing on theory is acceptable!
DIETARY RETINOIDS AND LIVER FIBROSIS
High, but not low, dietary retinoids aggravate manifestation of rat liver fibrosis
BRIGETTE VOLLMAR,* CHRISTINE HECKMANN,* SVEN RICHTER*, and MICHAEL D MENGER*
Background: Although there is strong implication that retinoids regulate Ito cell proliferation and collagen synthesis, results from in vivo studies on the relationship between vitamin A and liver fibrosis are conflicting. The present study focuses on the role of vitamin A in carbon tetrachloride (CCl4)-induced fibrosis by chronic feeding of rats with either a vitamin A-supplemented or -depleted diet.
Methods and results: In animals with high dietary hepatic retinoid levels, liver fibrosis was more pronounced and was associated with an increased CCl4-toxicity resulting in high mortality (73%). Enhanced liver fibrosis was confirmed by in vivo fluorescence microscopic determination of both collagen deposits (7.4 ± 1.1 vs 3.9 ± 0.3% in high vitamin A diet-fed and standard diet-fed fibrotic animals, respectively; P < 0.05) and rarefication of sinusoids (1.5 ± 0.2 vs 2.4 ± 0.2 sinusoids/200 µm in high vitamin A diet-fed and standard diet-fed fibrotic animals, respectively; P < 0.05). It was further associated with decreased bile flow and increased parenchymal cell damage. CCl4 reduced hepatic retinoid levels in high vitamin A diet-fed animals, but restored hepatic retinoid levels in animals fed with a vitamin A-deficient diet, implying major interference of vitamin A metabolism with hepatotoxic agents such as CCl4. Low vitamin A feeding did not modulate liver fibrogenesis and caused no mortality.
Conclusions: These results show that the vitamin A status of the liver plays an important role in liver fibrogenesis. While dietary vitamin A shortage does not promote liver fibrogenesis, high levels of vitamin A have the potential to increase systemic and hepatic toxicity of CCl4. Thus, the narrow therapeutic window for nutritional vitamin A substitution must take into account that liver fibrotic patients may display enhanced susceptibility to the adverse effects of vitamin A.
I found this thanks to MSC~
Summary In order to elucidate possible physiological roles of metallothionein (MT), we have studied immunohistological localization of MT in the eye of the rat, using an avidin-biotin peroxidase complex method. As a result, strong MT immunostaining was observed in the epithelium of the lens and cornea. In the retina, considerably strong MT immunostaining was observed in the pigment cell layer while the nerve fiber layer and inner plexiform layer showed weak MT staining. Glial cells in the optic nerve were found to have marked MT staining. The present result is consistent with the hypothesis that MT may be involved not only in activation of zinc enzymes and cell proliferation through supply of zinc ions, but also in a protective mechanism in the blood-retina barrier.
Now how do we get zinc in us??? I will tell you how....from those gd steel whiskers, that is how.
My god, they are coated with the zinc!!!!! I'm referring to those fake hairs. They call them whiskers and it is from steel. I kid you not. It is what your ac is made of too.
Thanks London and msc. I had two heavy metal tests done which both turned up highered levels of zinc. However, I worked at a zinc plant as a purchasing agent in my 20s and that site is now an EPA supersite and we attributed the elevated levels to my year spent rolling, yes rolling in the stuff. No one warned us how dangerous it was and it had very interesting properties. I also lived across the street from the plant and nothing would grow in the yard...I held my son in my work clothes and let him play in the yard. There was a settlement in the 80s or 90s but I was in California and did not know of it until I returned here to Okie in 92. My thought on the matter that after 30 years the zinc would have dissipated from my body so very interesting that I still have elevated zinc and other than elevated lead that was all these 2 tests found. Hhhhhmmm. Have to pull those records and have them looked at again with this in mind.
Thanks U 2.
During visual excitation, rhodopsin undergoes photoactivation and bleaches to opsin and all-trans-retinal1, 2. To regenerate rhodopsin and maintain normal visual sensitivity, the all-trans isomer must be metabolized and reisomerized to produce the chromophore 11-cis-retinal in biochemical steps that constitute the visual cycle and involve the retinal pigment epithelium (RPE; refs. 3, 4, 5, 6, 7, . A key step in the visual cycle is isomerization of an all-trans retinoid to 11-cis-retinol in the RPE (refs. 9, 10, 11). It could be that the retinochrome-like opsins, peropsin, or the retinal G protein-coupled receptor (RGR) opsin12−16 are isomerases in the RPE. In contrast to visual pigments, RGR is bound predominantly to endogenous all-trans-retinal, and irradiation of RGR in vitro results in stereospecific conversion of the bound all-trans isomer to 11-cis-retinal17. Here we show that RGR is involved in the formation of 11-cis-retinal in mice and functions in a light-dependent pathway of the rod visual cycle. Mutations in the human gene encoding RGR are associated with retinitis pigmentosa18.
hard to understand, I know, but here is the rest of the article....
http://www.nature.com/ng/journal/v28/n3 ... 1_256.html
I found this by googling: retinyl esters and fibre opticle
Mi cuerpo tienes muchos gracias para te
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