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The Fiber Disease

Human Anatomy, Physiology, and Medicine. Anything human!

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Postby Cilla » Wed May 17, 2006 7:58 pm

Randy,

If Tam tam were being untruthful, he would not wish the NIH contacted, on the basis of his claims, seeking ultimately a criminal investigation.

If they proceed with this, they will contact him. They are consummate professionals.

Write to them, Barz. You do not have to say anything here regarding this if you do not want to.
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Postby London » Wed May 17, 2006 8:19 pm

Well, well., well....

This is all I'm saying about this so don't even ask me for more info.

As some of you might know ( I just saw it yesterday evening) there is

a new blogger that is ripping up Morgellons Foundation and Ginger and

us the victims....... ( do not know the website and am not going back there

to visit it anyway) but what I do know is that it had a poster on there

by the name of Slim ????something.......WEll. I know WHO THAT PERSON

IS AND THEY USED TO WRITE ( A LOT) ON THIS FIBER FORUM. Oh,

what a shame......and I know that they are reading this now and thinking how the hell did she find out.........

Let me tell you ( female poster that is a fake and a Fed) you are being watched
just like your watching us. And something else.....I told you this- that we

have our ways of watching too..........Have a good day people and beware........
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Postby London » Wed May 17, 2006 8:37 pm

A fiber optic biosensor (FOBS) to monitor mutans streptococci in human saliva.

Kishen A, John MS, Lim CS, Asundi A.

Biomedical Engineering Research Center, Nanyang Avenue, Nanyang Technological University, Singapore 639798, Singapore. ckishen@ntu.edu.sg

A fiber optic biosensor (FOBS) to monitor mutans streptococci activity in human saliva is developed.


The biosensor utilizes e fiber optic evanescent wave spectroscopy to monitor a bacterial mediated biochemical reaction. To achieve this, a short length of the cladding is removed; the fiber core surface is treated and coated with a thin film of porous glass medium using sol-gel technique. The mutans streptococci mediated reaction with sucrose is monitored using a photosensitive indicator, which is immobilized within the porous glass coating. Spectroscopic analysis shows that the transmitted intensity at 597 nm increases conspicuously when monitored for 120 min. Two distinct phases are observed, one from 0 to 60 min and the other from 60 to 120 min. A negative correlation coefficient between the rate of increase in absorption peak intensity recorded by the FOBS and the decrease in pH measured using the pH meter, was calculated to be rho=-0.994. This investigation highlights the potential benefits of this sensor to monitor mutans streptococci activity in saliva.
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Postby London » Wed May 17, 2006 8:54 pm

and Skytroll,

You will love this one. It is on Post -doc information in US institutions.

http://www.nap.edu/books/0309069963/html/
London
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Postby London » Wed May 17, 2006 9:22 pm

and one time this person posted a bogus phone number that is no longer listed.......but I have it> you see you ______girl, I saved every post since day one,,,,,,you aint there.....

you don't live in the state you say you do, in fact your way, way way away from that state.......

and again, the phone number is not happening......Now, I'm not going to say your name, but cross me just one time or hurt my friend Carrie just one time and I will rip you to shreads.....and oh yeah, I know I will be banned if I do it here....and that is what I will deal with.....so if I were you I would quit with my blogging girlfriend.....

Like I said....you drop it and so will I.
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Re: What?

Postby ukguy » Wed May 17, 2006 10:56 pm

RANDY wrote:
This child is sick and it is killing this parent to see this. Saying a video is coming up is just horrible...it will not give thsi parent any relief and Tam not answering this parent is just EVIL.

Randy


Third


Randy,

Don't you think if TamTam could help RIGHT now with an answer he would?

I'm getting sick and tired of your animosity and continued character
assassinations. You've said many times that you've had it with this
board, presumably because you were not getting your own way.

Your way appears to be controlling the opinions of everyone here
until you are happy everyone thinks along the same lines as you.

Ok, and what is your belief about this disease?

You have no idea.

Ok, so for the people interested in finding an answer...let them continue
here and excercise their own judgement without having the thread
ruined every few days with your insults.

If your answer lies elsewhere Randy then please...follow your instincts.

Thanks,
UKguy
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HONESTY

Postby RANDY » Wed May 17, 2006 11:37 pm

Cilla wrote:Randy,

If Tam tam were being untruthful, he would not wish the NIH contacted, on the basis of his claims, seeking ultimately a criminal investigation. If they proceed with this, they will contact him. They are consummate professionals.

Write to them, Barz. You do not have to say anything here regarding this if you do not want to.


WISH THE NIH TO BE CONTACTED?...more bull. Criminal investigation? About what? Nothing has been proven! This is fairy land perpetuated.

Elias Zehoui.there is a name for you .....tell him you have the disease mentioned by Randy Yaskal and her friend Djamel the head of the Algerian Chamber of Commerce. He will tell you what he told me and Djamel when we asked to have lunch with him to discuss this disease. He told Djamel......what I have been telling you over and over again.


I will go check if anyone calls him. If they do they will get the same answer I did.

Anyways......After being told what I was told by my buddies and getting no repsonse from Tam or anyone about talking to them I would never bring his findings to anyone of importance cuz I would look like a fool and a crazy person...which I am starting to realize is the goal of this group who keeps on insisting that we do this. I would never go forward with this video unless I had a scientist talk to Tam...NEVER GONNA HAPPEN!

I doubt anyone is going to contact the NIH. And anyways it does not work the way you are stating. It works one way and only one way. You are misleading everyone stating that he is going to do anything.

I am telling the truth here..so shoot me. Very strange that the only person speaking the truth here is getting shot in the butt.

Interesting. But I do not care about me. I care about the disease. So I will continue to speak my mind.

For the millionth time:(Mark my words and you will see I am correct.)

You get a University to apply for a grant, they do the research, you come to a conclusion or apply to continue research. You write a paper. The NIH has a review board. They read the paper. You get peer review and you publish the paper. The NIH alerts the CDC and you have a new disease.
THAT IS THE ONLY WAY TO GET THIS DONE.

You can not tell the NIH what Tams states..nothing has been discovered or proven yet. Good luck. I bet everyone $20.00 right now. NOTHING IS GONNA HAPPEN.

Alerting them to what? Nothing has been discovered that has been proven.

This advice only leads to us looking crazy and make a good headline or an episode for a movie script. Not at our expense.

Tam is gonna get WHO???? to do what??????? regarding saying something exists????? You gotta prove it exist by following the above procedure.

I am telling the truth here. Why do you not want to hear it? That is really weird.

I could care less if anyone likes or dislikes me. I am here for one reason to tell the truth and protect those from false words.

My heavens Barz is begging..no one should ever have to beg for their child to get someone to help....that is horrible!

And if Tam was decent, and I PM'd him about this... he would have written that his advice on meds came directly from the mouth of Dr Schwartz..I guy I FOUND. (And Jeff told me about....credit to Jeff.) It was not Tam's idea at all. So sad. Sure sounded like he thought of it. He should have credited Schwartz.

End of story!


Randy

Sorry Poison. And the best advice you get get is to treat the bacterial, fungal and parastic in that order. It is up to you but that is the protocol that works along with the other things I have said. I have had this monster for over 4 years. I can work a 40 hour work week without getting tired. That is a success story. Also get all your blood work monitored and get a CT scan if you can. This causes inside and outside lesions. These are proven truths. You have to battle this on three fronts.
The longer you take to do that once this hits you the sicker you will be in the years to come. HONEST!
During the End Times, Good will battle Evil. Where do you stand?
http://unknownskindisease.com
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Postby ukguy » Thu May 18, 2006 12:23 am

Randy

I actually agree with alot of what you say and at the end of the
day you're entitled to your opinion but surely you can see that
there has to be a better way to voice it?

Thanks
Ukguy
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Postby RANDY » Thu May 18, 2006 1:48 am

I am a Jewish American Princess from Long Island who had lots of money growing up and is poverty sticken now..due to thsi ailment.....with an IQ one point BELOW..AHHHHHHHH! Mensa acceptance that raised a son as a single mom with no child suppport or alimony and who has had this disease for a long time and now has her mother, who just went through a lumpectomy, living with her..AHHHHHH!!! who also has a brother with diabetes since he was 3 at risk of losing both legs.... and a dad that died of Alzheimers..so sugar coating things has never really been my best quality. I am kinda raw. Sorry!
During the End Times, Good will battle Evil. Where do you stand?
http://unknownskindisease.com
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Postby London » Thu May 18, 2006 2:07 am

Well besides the sea lamprey and the hagfish I think make up our lesions,

I have been really looking at this: Pfiesteria. It is invading all of our oceans and has human documented cases even.

The following is a snippet I found on E. Coli:
___________________________________

Among 216 commensal E. coli strains we found 27 mutators (12.5%). 9/69
mutators (13%) were observed among strains isolated from Dogons, an
isolated African tribe, which is not substantially different from the
15/84 isolated in Croatia (18%) and 3/63 observed among commensal strains
isolated in Paris (5%), nor from the 42/288 found among pathogenic
isolates (15%). Among pathogens, the mutators were distributed as follows:
7/61 strains from neonatal meningitis (11%), 21/145 from urinary tract
infections (14%), 3/16 from bacteremia (15%), 1/14 from miscellaneous
infections (7%), and 10/52 from strains causing haemolytic-uremic syndrome
or haemorrhagic diarrhea (19%).
9. The pathogenic E. coli strains must cope with host's defense
mechanisms and with nutrient-limited environment during infection, while
the commensal strains must cope with selective pressures exerted by the
other inhabitants of the gut microflora and probably to a lesser extent
with the host's immune response. Thus, pathogenicity represents only one
form of bacterial specialization to a particular niche [(1); E. A.
Groisman and H. Ochman, Trends Microbiol. 2, 289 (1994) [Medline]].
10. F. Taddei et al. Nature 387, 700 (1991).
11. L. Chao and E. C. Cox, Evolution 37, 125 (1983) ; W. Trbner and R.
Piechocki, Z. Allg. Mikrobiol. 21, 347 (1981) [Medline]; Mol. Gen. Genet.
198, 175 (1984); Naturwissenschaften 72, 377 (1985); L. Chao, C. Vargas,
B. B. Spear, E. C. Cox, Nature 303, 633 (1983) [Medline].
12. Strains isolated in a university hospital (Paris, France) showed 15/67
mutators (22%), which has to be compared with 3/58 in a general hospital
(Avignon, France) (5%) and 2/19 in a general practitioner's laboratory
(Paris, France) (11%). However, the B carboxylesterase typing showed
similar genetic heterogeneity in these three populations (B. Picard,
unpublished data). Similarly, comparison by extended ribotyping of
Pseudomonas cepacia strains isolated from cystic fibrosis patients showed
that strains responsible for the acute infections had a stronger
instability than those associated with chronic infections [ K. R. Rozee,
D. Haase, N. E. MacDonald, W. M. Johnson, Diagn. Microbiol. Infect. Dis.
20, 181 (1994) [Medline]]. Furthermore, in the different Yersinia species
increased virulence correlates with increased genetic instability [ A.
Guiyoule, et al., J. Clin. Microbiol. 32, 634 (1994) [Medline]; H.
Najdenski, I. Iteman, E. Carniel, Contrib. Microbiol. Immunol. 13, 281
(1995) [Medline];
London
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Postby London » Thu May 18, 2006 2:27 am

You want to see what's wrong with our whole ecolgical world?

Check out these titles and it list the journals they are in. It tells everything from what fugi exist on the bottom of the ocean floor to the timber........

and......also, this worm thing is in some types of paper........that's right!

I think it is Japenese writing paper-

Here's the document on Ecology-

http://www1.elsevier.com/homepage/sah/s ... 91caes.pdf
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Postby London » Thu May 18, 2006 2:37 am

and I have your lepidoptera right here; right here along with BT poisoning............


Analysis of midgut proteinases from Bacillus thuringiensis susceptible and resistant Heliothis virescens (Lepidoptera: Noctuidae)

Departments of Entomology1, Biochemistry and Molecular Biology2, University of Georgia, Athens, GA-30602. USDA ARS Grain Marketing and Production Research Center3, Manhattan, KS-66502.
Heliothis virescens is a major lepidopteran pest of cotton in the United States and the target insect of Bacillus thuringeinsis (Bt) transgenic cotton. We conducted an analysis of gut proteases from Bt susceptible and resistant H.virescens strains, including the susceptible strain, YDK, and three resistant strains, YHD2-B, CXC, and KCBhyb. Casein zymogram analysis of YDK and YHD2-B gut extracts did not reveal significant differences. However, two unique bands of caseinolytic activity were observed in CXC and KCBhyb. Kinetic microplate assays with a trypsin substrate demonstrated that proteinases in YDK gut extract had more alkaline pH optima compared to YHD2-B, CXC and KCBhyb. In assays with a chymotrypsin substrate, enzymes in YDK extracts had lower alkaline pH optima in contrast to those in YHD2-B gut extract. Enzymes from KCBhyb gut extracts had the highest activity of all strains in alkaline buffers, particularly at pH 10.6, similar to the physiological pH of the lepidopteran midgut. Temporal Cry1Ac protoxin activation indicated that YHD2-B gut extract processed protoxin at a slower rate than that of YDK. Because gut proteinases are a critical component of Bt toxin mode of action, these differences may contribute to decreased toxicity in the Bt-resistant strains.

http://www.ent.iastate.edu/sip/2005/node/272

and:

The evolution of virulence and transmission of disease
Philip Agnew
Génétique et Evolution des Maladies Infectieuses, CNRS / IRD - UMR 2724, 911 Avenue Agropolis (bp 64501), 34394 Montpellier Cedex 05, France.
A pathogen's virulence is an important trait for anyone concerned with pathogens or the host populations they attack. It is also a trait often related to a pathogen's transmission success. The ability to predict or eventually manage the evolution of a pathogen's virulence is a highly desirable goal. Many theoretical models have been developed with this in mind. The aim of my talk will be to outline how these models are constructed, the trade-offs involved, and how predictions are made. Although much of the data required for such models is generated during studies of invertebrate pathology, I will highlight data that are not routinely collected but could be and that would improve the quality of model predictions. I will also indicate where the development of in theory is currently hampered for want of appropriate data and to which invertebrate pathologists could make a valuable contribution.
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