Human Anatomy, Physiology, and Medicine. Anything human!
Gee I really need to proof things before I send them out. Alias..not alian....pieces not peaces....the list goes on. Please try to stuggle with all the typos.
I must be tired.
OH and these biofilms mobsters need only a quorom to decide to mutate and be resistant to antibiotics and by forming in gangs they can evade being singled out and prosecuted by the medication brigade and when they feel like it they can incorporate other gangs of different bacteria into their crib by a minimum vote of the gang members causing a really scary gang that no one wants to take down.
Have I go this right?
Thank you for your reply. I meant to address my question to "Your mother" Sorry about that.
Cell signalling and Trypanosoma cruzi invasion
Burleigh BA, Woolsey AM.
Department of Immunology and Infectious Diseases, Harvard School of Public Health, 665 Huntington Ave, Bldg I Rm 713, Boston, MA 02115, USA. [email protected]
Mammalian cell invasion by the protozoan pathogen Trypanosoma cruzi is critical to its survival in the host. To promote its entry into a wide variety of non-professional phagocytic cells, infective trypomastigotes exploit an arsenal of heterogenous surface glycoproteins, secreted proteases and signalling agonists to actively manipulate multiple host cell signalling pathways. Signals initiated in the parasite upon contact with mammalian cells also function as critical regulators of the invasion process. Whereas the full spectrum of cellular responses modulated by T. cruzi is not yet known, mounting evidence suggests that these pathways impinge on a number of cellular processes, in particular the ubiquitous wound-repair mechanism exploited for lysosome-mediated parasite entry. Furthermore, differential engagement of host cell signalling pathways in a cell type-specific manner and modulation of host cell gene expression by T. cruzi are becoming recognized as essential determinants of infectivity and intracellular survival by this pathogen.
Recombinational Repair of DNA Damage in Escherichia coli and Bacteriophage λ
http://www.pubmedcentral.com/articleren ... rtid=98976
Great find London.....we really need a conferencing device so we can figure out how to put all our new found knowledge into one huge paper that is easy to understand.
Now this comment came from my Yale Physics/Engineering buddy and is not meant to be rude or snotty. It is just his honest opinion when he reviewed all that Tam wrote divided by the threads he placed them in.
For what it's worth, I am a firm believer that any self-proclaimed specialist in any field of human endeavor should be able to explain what he does to a 5th-grader in 10 sentences or less, and if he can't, then he is a charlatan. I can explain Einstein's theory of relativity to you in 10 sentences or less, since it is my field. TamTam did not make any sense in a hundred of sentences he wrote. By that yardstick he is a charlatan.
Sorry I missed you yesterday! Too tired. I woke up at 10 pm last
night. I'm talking slept ALL day.
Did you see how long that second article I posted was? Whew. I'm hoping some Dr., pharmacist or a chemist will read it and then explain
it to us.
I read where you said something about them trying to make us sterile.
Could you explain more of this? Or maybe it's there in your post and I just need to go back and re-read it. but if not, could you please post why you think that.
Here's my take on it ( right now anyway) . I think they are trying to find a way to halt cervical cancer.
I really do. I think this wholle damn thing is about them
making a novel ( new Drug). Maybe Tam is right. I think when he told me:
Steps, Steps, Steps.........
that he was talking about the chemicle steps involved in the process of
making a drug.
Yep, an anti-cancer drug. I Don't know for sure but maybe our old friend
TamTam would enlighten me. Or does he even know?
TAMTAM, PLEASE ADDRESS!
WHERE oh WHERE is YYZ? yyz, you okay??? Please write us.
Also Randy, or ANYONE else, what do you think about:
Mason-Pfizer monkey virus (MPMV) ???
and here's another good one:
Suppressor of Cytokine Signaling (SOCS) Proteins Indirectly Regulate Toll-like Receptor Signaling in Innate Immune Cells*
also note this:
Organelle not in animal cells
The organelle is found in prokaryotic cells but not eukaryotic cells of animal origin.
and look at this one: PLEASE, SOMEONE LET ME KNOW WHAT THEY THINK ....mAYBE THEY ARE TRYING TO MAKE ANOTHER WHOLE kINDOM!!!
"The Production and Directed Differentiation of Human Embryonic Stem Cells"
http://edrv.endojournals.org/cgi/conten ... 005-0016v1
AND NOW OLDE TAMY NEEDS TO SEE THIS:
Biodegradation of Two-Component Polymer Mixtures
M. Ratajska, S. Boryniec, A. Wilczek, M. Szadkowski
Institute of Chemical Fibres
ul. M. Sklodowskiej-Curie 19/27, 90570 lódz, Poland
Synthetic films filled with natural components were biodegraded in an aqueous medium and in soil. Samples were removed and examined after 3, 6 and 9 months of biodegradation. The changes which took place in film material were studied gravimetrically, by electron microscopy and water absorption measurements. As a result of the studies it was found that the changes in two-component material caused by biodegradation were more associated with the type and form of the natural component than with the synthetic component. Application of the coarse-grained fillers caused damage in the form of less numerous but larger changes. When a fine-grain filler was used, the changes in the polymeric material were numerous, small and scattered. Especially the method based on water-absorbing capacity perfectly visualizes the changes in the film material.
Key words: biodegradation, two-component polymer mixtures, micro-organisms, synthetic polymers, natural polymers.
One more thing London. You gotta start with what we all have in common which are the fibers and work it from there. So many things are interconnected in the body that you can pull stuff off the internet for days.
Start and end with the fibers if you can. I will try and do the same. I , like you have only so many hours in a day to read and read and read.
Right now I am followinf Tams path of words to see if they make sense when I break them down or if he is just playing G_d and the Devil with his made up society of peasnats with illnesses for which he ahs chosen us because he looked up the name Morgellons and the word BLACK MORGELLONS is on that site of the Shadowdancers.
Lucky google for him and his game.
But maybe he is true so I will spend my only day off figuring it out..when I have a hosue to clean and errands to run.
All we have to blame is CDC who ignores the urgency of MORGELLONS' sufferers "crying for healing", not accusing TamTam's posting as "I have taken notice of your postings via VIS hardware and due to the nature of your methodology I can not help but think of it as an attempt to release data of a sensative nature that has been gleened from work done in a closed area possibly involving a threat appendix specifically the survivability segment.
By releasing all the information presumed to be in the public domain and not releasing information of a proprietory nature "Cleaning it up" can be difficult at best. This I understand, however if there is any way you can expedite this process I am certain it would be greatly appreciated by all affected."
We must urge US Congress for the fund to do more research on Morgellons. TamTam alone can not help too much in the 'CURE' of this notorious SUPERBUG.
http://ift.confex.com/ift/2005/techprog ... _28472.htm
Effect of quorum sensing inhibitors on food spoilage
M. RASCH1, T. B. Rasmussen2, T. O. Larsen2, M. Givskov2, and L. Gram1. (1) Dept. of Seafood
Research, Danish Institute for Fisheries Research, c/o Technical Univ. of Denmark, Building 221,
DK-2800 Kgs. Lyngby, Denmark, (2) BioCentrum, Technical Univ. of Denmark, DK-2800 Kgs. Lyngby,
Many Gram-negative bacteria use acylated homoserine lactones (AHLs) to regulate expression of
phenotypes in relation to cell density, the so-called quorum sensing (QS). QS is involved in
expression of virulence and spoilage in a number of Gram-negative bacteria. AHLs have been
detected in several types of spoiling food products. Bacterial spoilage of bean sprouts is
characterized by soft rot and the most important bacteria are pectinolytic Erwinia carotovora
and Pseudomonas fluorescens. QS is employed to regulate spoilage phenotypes such as protease,
pectinase, cellulase, and siderophore production in a bean sprout spoiling Pectobacterium. Also,
clotting of milk by Pectobacterium is regulated by QS. Consequently, compounds that block QS
(quorum sensing inhibitors (QSI)) without affecting growth of the bacteria might become
instrumental in novel food preservation strategies. Halogenated furanones from the red alga
Delisea pulchra share structural similarities with AHLs and inhibit a number of QS systems.
Chemical reactivity and instability as well as toxicity of some compounds stresses the need for
new QSI compounds. We have evaluated the effect of a synthetic analogue of the D. pulchra
furanones, furanone C-30, and a series of new QSIs on the expression of spoilage in two food
model systems where spoilage is controlled by bacterial QS: bean sprouts and milk. Some of the
QSIs, which were used in concentrations where bacterial growth was not inhibited, reduced
protease, pectinase and siderophore production of the bean sprout spoilage Pectobacterium. In
three of five experiments, bean sprouts inoculated with the food spoilage Pectobacterium and
treated with C-30 spoiled later and to a lesser extent than non-treated sprouts. Treatment with
several different new QSIs delayed the clotting spoilage of milk. Results from these experiments
as well as the perspectives of using QSIs to delay food spoilage will be discussed.
Session 42, Control of quorum sensing signals and antimicrobial effects on foods
2:30 PM - 5:30 PM, Monday PM Room 296
2005 IFT Annual Meeting, July 15-20 - New Orleans, Louisiana
Last edited by J Jill on Sat Jan 28, 2006 6:52 pm, edited 1 time in total.
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