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Senescence & Cancer

Discussion of all aspects of cellular structure, physiology and communication.

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Postby DevGrp » Mon Feb 21, 2005 12:08 pm

I have researched the genetics of fungal lipid and sterol biosynthesis, plant pathology and fungicide resistance, diabetic neuropathy, endocrinology of breast and endometrial cancer (including work on angiogenesis), endocrinology of diabetes and heart disease (more blood vessel work), commercial applications of plant lipases. Am currently working on the strain improvement of potential non food crops.
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Postby thank.darwin » Thu Feb 24, 2005 1:19 pm

Wow, you've done a lot :o
No amount of experimentation can ever prove me right; a single experiment can prove me wrong.
-Albert Einstein
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Wow!!

Postby 2810712 » Fri Feb 25, 2005 11:17 am

Wow!!
Just fantastic!

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Postby Poison » Fri Feb 25, 2005 3:58 pm

woow!!! I think so. You've really done a lot. :)
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Postby jules_dgreat! » Fri Feb 25, 2005 7:35 pm

There has been no medical reports suggesting that cancer cells, or normal cells who have gone awry in their cell division process thereby producing cancer cells undergone further changes to produce normal cells. Basically, cancer cells are mutated normal cells and possibility of becoming normal is doubtful.
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Postby mith » Fri Feb 25, 2005 8:29 pm

because the odds of getting any base pair mutated is better than getting a specific mutated base pair to mutate back.
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Postby jules_dgreat! » Fri Feb 25, 2005 9:01 pm

That is correct. :)
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Postby brspurri » Wed Aug 10, 2005 7:18 pm

Cancer cells reproduce the same as a normal cell...copying that cells DNA and splitting. The cancer cell has contains mutated DNA (from the original cell), and thus copies the mutated DNA as well. Since it takes 4-5 mutations for a normal cell to become malignant, the cancer cell would have to mutate in EXACT OPPOSITION to the previous mutations in order to produce a complete normal cell. This is highly improbable. Hope this makes sense.
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Postby chemistry_freako » Thu Aug 11, 2005 12:29 am

hmms - dunno if there's any link - but senescence can be due to the shortening of the repeats at the telomeres of chromosomes right?
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Postby Poison » Thu Aug 11, 2005 7:09 pm

It has relation with telomere shortening. Senescence means 'aging'. Which is not seen in cancer cells. If you stimulate that, the cell will some day 'die'. THis will be (I hope so) a good way of threating cancer. But how to stimulate aging? thats the main problem.
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Postby chemistry_freako » Fri Aug 12, 2005 3:40 am

stimulate ageing? =\
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Postby Poison » Fri Aug 12, 2005 6:10 pm

Cancer cells do not go through apoptosis. Is it clear now?
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