Discussion of all aspects of cellular structure, physiology and communication.
I have researched the genetics of fungal lipid and sterol biosynthesis, plant pathology and fungicide resistance, diabetic neuropathy, endocrinology of breast and endometrial cancer (including work on angiogenesis), endocrinology of diabetes and heart disease (more blood vessel work), commercial applications of plant lipases. Am currently working on the strain improvement of potential non food crops.
There has been no medical reports suggesting that cancer cells, or normal cells who have gone awry in their cell division process thereby producing cancer cells undergone further changes to produce normal cells. Basically, cancer cells are mutated normal cells and possibility of becoming normal is doubtful.
because the odds of getting any base pair mutated is better than getting a specific mutated base pair to mutate back.
Living one day at a time;
Enjoying one moment at a time;
Accepting hardships as the pathway to peace;
Cancer cells reproduce the same as a normal cell...copying that cells DNA and splitting. The cancer cell has contains mutated DNA (from the original cell), and thus copies the mutated DNA as well. Since it takes 4-5 mutations for a normal cell to become malignant, the cancer cell would have to mutate in EXACT OPPOSITION to the previous mutations in order to produce a complete normal cell. This is highly improbable. Hope this makes sense.
It has relation with telomere shortening. Senescence means 'aging'. Which is not seen in cancer cells. If you stimulate that, the cell will some day 'die'. THis will be (I hope so) a good way of threating cancer. But how to stimulate aging? thats the main problem.
It matters not how strait the gate
How charged with punishment the scroll
I am the Master of my fate
I am the Captain of my soul.
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